Suchergebnisse

Previous research on the Repression-Sensitization (R-S) Scale found that repressors gave fewer sexual associations than sensitizers on a double entendre word association task. That difference, however, may have been due either to differential familiarity, or external, social inhibition,
rather than to internal, cognitive defense processes. In the present experiment, male sensitizers, neutrals, and repressors performed an identification task and an association task with word lists containing double entendres. They received one of three instruction sets, designed either to
facilitate, inhibit, or disregard the possible sexual associations. The identification task results failed to support the familiarity hypothesis. The association task results suggested that repressors are more responsive to external, social inhibition cues than are either sensitizers or neutrals.

AbstractInhibitory control (IC) is a core executive function integral to self‐regulation and cognitive control, yet is itself multi‐componential. Directed global inhibition entails stopping an action on demand. Competitive inhibition is engaged when an alternative response must also be produced. Related, but not an executive function, is temperamentally‐driven wariness of novelty, known as behavioural inhibition. Understanding early development of these components has been hampered by a shortage of suitable measures. We combine established and novel measures to capture directed global inhibition (Toy Prohibition, Touchscreen Prohibition), competitive inhibition (A‐not‐B, Early Childhood Inhibitory Touchscreen Task; ECITT) and behavioural inhibition (Touchscreen Approach) in 113 10‐ and 16‐month‐olds (73 seen longitudinally). ECITT performance shows good 1‐week test‐retest reliability at 10‐months (r = 0.30–0.60) but little stability to 16‐months. Directed global inhibition performance shows developmental progression but little stability of individual differences from 10 to 16 months. Performance on measures targeting similar IC components shows greater coherence at 16‐months (r = 0.23–0.59) compared with 10‐months (r = 0.09–0.35). Probing of ECITT condition effects indicates toddlers are more able, compared with infants, to override immediate prepotencies; indicative of increasingly flexible control over behaviour. However, exerting IC over cumulative prepotencies appears just as challenging for toddlers as infants. Exploratory analyses show little evidence for cross‐sectional or longitudinal associations between behavioural, directed global and competitive inhibition. In combination, these findings indicate that IC is not yet a stable, unidimensional construct during the transition between infancy and toddlerhood, and highlight the need for careful selection of multiple measures for those interested in capturing early variation in IC.

1Inhibition of Chemical Carcinogenesis by Phenols, Coumarins, Aromatic Isothiocyanates, Flavones, and Indoles -- I. Introduction -- II. Inhibitors of Chemical Carcinogens -- III. Discussion -- 2Inhibition of Carcinogen Metabolism and Action by Disulfiram, Pyrazole, and Related Compounds -- I. Introduction -- II. Disulfiram -- III. Sodium Diethyldithiocarbamate and Dithiocarbamate Pesticides -- IV. Carbon Disulfide -- V. Pyrazole -- VI. Polycyclic Aromatic Hydrocarbons: Benzo[a]pyrene and 7,12-Dimethylbenz[a]anthracene -- VII. Hydrazo and Azoxy Carcinogens -- VIII. N-Nitrosamines -- IX. Arylamines -- X. Azo Dyes: 3?-Methyl-4-dimethylaminoazobenzene -- XI. Ultraviolet Light -- XII. Spontaneous Tumors -- XIII. Other Effects of Thiono Sulfur Compounds -- References -- 3Retinoids and Chemoprevention of Cancer -- I. Introduction -- II. Retinoids and Epithelial Cell Differentiation -- III. Suppression of Malignant Transformation and Tumor Promotion by Retinoids -- IV. Retinoid Deficiency and Carcinogenesis -- V. Natural Retinoids and Prevention of Carcinogenesis -- VI. Structure-Activity Relationships of New Synthetic Retinoids -- VII. Prevention of Cancer in Experimental Animals with New Synthetic Retinoids -- VIII. Mechanism of Action of Retinoids in Chemoprevention of Cancer -- IX. Mechanism of Toxicity of Retinoids -- X. Combination Chemoprevention with Retinoids -- References -- 4Ascorbic Acid Inhibition of N-Nitroso Compound Formation in Chemical, Food, and Biological Systems -- I. Introduction -- II. In Vitro Studies -- III. In Vivo Studies -- IV Tests on Carcinogenicity and Mutagenicity of Ascorbic Acid -- V. Effects of Ascorbic Acid on Carcinogenicity and Mutagenicity of N-Nitroso Compounds -- VI. Ascorbic Acid and Carcinogenesis in Man -- VII. Summary and Conclusions -- References -- 5?-Tocopherol (Vitamin E) and Its Relationship to Tumor Induction and Development -- I. Introduction -- II. Vitamin E as an Antitumor Agent -- III. Nitroso Compounds -- IV. Formation of N-Nitroso Compounds -- V. Blocking N-Nitroso Compound Formation -- VI. ?-Tocopherol Applications -- VII. Ascorbic Acid and Tocopherol Effect on Preformed Nitrosamines -- VIII. Summary -- References -- 6Trace Elements and Metals as Anticarcinogens -- I. Introduction -- II. Selenium -- III. Zinc -- IV. Copper -- V. Other Trace Elements and Metals -- VI. Closing Remarks -- References -- 7Plant Sterols: Protective Role in Chemical Carcinogenesis -- I. Background -- II. Plant Sterols: Structure and Function -- III. Animal Test Systems -- IV. Results -- V. Discussion -- References -- 8Immunoprevention -- I. Introduction -- II. Detection of Tumor-Associated Antigens in Experimental Rat Bowel Carcinomas -- III. Evidence that Embryonic Antigens are Associated with Bowel Carcinomas -- IV. Enhanced 1,2-Dimethylhydrazine-Induced Tumorigenesis in Immunosuppressed Rats -- V. Inhibitory Effect on Bowel Carcinogenesis by Immunization with Transplantable Syngeneic Colon Carcinoma -- VI. Inhibitory Effect on Bowel Carcinogenesis by Immunization with Fetal Tissue -- VII. Inhibition of 1,2-Dimethylhydrazine-Induced Carcinogenesis in Multiparous Rats -- VIII. Effect of Tumor Resection on the Development of Additional Primary Tumors -- IX. Regression of Early Primary Bowel Carcinomas by Multimodal Immunological Treatment -- X. Conclusions -- References -- 9Summation and Future Challenges -- I. Introduction -- II. Challenges to Chemists and Molecular and Cell Biologists -- III. Challenges to Epidemiologists and Oncologists.

Emerson may not have been what is called a political philosopher, but political considerations played a more subtle part in his thinking than mere expressions of opinion on public affairs would suggest. (SJK)

Abstract
Oxidation of fats and oils reduces the nutritional value of food and causes various health problems. The addition of antioxidants prevents the oxidation of fats in the food. The antioxidant activity of antioxidants represents the ability to inhibit the process of oxidation. The antioxidant activity of herbal extracts has usually been assessed in a linoleic acid emulsion system. The aim of the present study was to evaluate inhibition of conjugated diene formation in Latvian linseed oil. Ethanol extract of the calyx of Hibiscus sabdariffa L., vanillin, α-tocopherol and 2,6-di-tert-butyl-4-methylphenol as additives were compared for their antioxidative activity. The samples of linseed oil with additives were incubated for 24 h at 60 °C and then analysed using UV spectrophotometry (λ = 234 nm). The antioxidant activity of additives was characterised by the percentage of formation of conjugated dienes. The ability of additives to inhibit oxidation in linseed oil decreases as follows: vanillin, 2,6-di-tert-butyl-4-methylphenol and α-tocopherol. Our results indicate that linseed oil can be used to test antioxidative activity of substances.