Suchergebnisse

Suicide claims approximately one million lives worldwide each year, but it is increasingly recognized that there are ways in which some of this loss of life can be prevented. Part of the Oxford Psychiatry Library, the second edition of Suicide Prevention places suicide in an historical and contemporaneous context, noting how interpretations of its causes and prevention have changed over the years. This comprehensive but concise pocketbook provideshealthcare professionals with an appreciation of the subtle relationship between illness and biological factors, and their interaction with society

Intro -- Title Page -- Copyright Page -- Contents -- Foreword -- Preface -- Acknowledgments -- Introduction -- Abbreviations -- Part I: Defining Readmissions and their Impact on the Healthcare Delivery System -- 1. The Affordable Care Act and the Readmission Problem -- Hospital Readmission: A Practical Definition -- Historical Background of the Readmission Problem -- What Causes Hospital Readmissions? -- All-Cause Readmissions -- The Readmission Problem -- Readmission Trends 2010 to 2013 -- Tactical Approaches to the Readmission Problem -- 2. Moving Beyond the Gold Standard -- The "Putting Heads in Beds" Mind-Set -- The Problem of Observation Days -- Collecting Readmission Data -- No Cookie-Cutter Answers -- Perspective: The Impact of ICD-9-CM and ICD-10 on Readmission Measurement under the ACA -- Part II: The Three Phases of Preparation to Prevent Readmissions -- 3. Prevention Planning Phase One: Before Discharge -- Conduct Patient Risk Assessments -- Create a Post-Acute Care Network -- Narrow the Hospital's Provider Network -- Align Contracts with Post-Acute Care Network Members -- Implement a Transitional Care Program -- Promote Patient Choice and Adhere to Antisteering Regulations -- Form Community Coalitions -- Develop a Secondary-Market Post-Acute Care Provider List -- Encourage Patient Self-Management -- Promote Patient Health Literacy and use Teach-Back Methods -- Collect Facility-Specific Data -- 4. Prevention Planning Phase Two: After Discharge -- Perform a Medication Reconciliation -- Schedule Post-Acute Care Follow-up Visits -- Set up Home Delivery of Medications -- Establish an Ambulatory Case Management System -- Provide Remote Monitoring for the Patient -- Post-Acute Care Network Tactics -- 5. Prevention Planning Phase Three: The Patient Returns to the Emergency Department -- Train Medicare Reimbursement Experts.

Intro -- CONTENTS -- LIST OF PAPERS -- LIST OF CONTRIBUTORS -- LIST OF TABLES -- Introduction -- 1. Famine Prevention in India -- 2. Famine Prevention in Africa: Some Experiences and Lessons -- 3. Ethiopian Famines 1973-1985: A Case-Study -- 4. Modelling an Early Warning System for Famines -- 5. Market Responses to Anti-hunger Policies: Effects on Wages, Prices and Employment -- 6. The Food Crisis in Africa: A Comparative Structural Analysis -- INDEX -- A -- B -- C -- D -- E -- F -- G -- H -- I -- J -- K -- L -- M -- N -- O -- P -- Q -- R -- S -- T -- U -- V -- W -- Y -- Z -- SUBJECT INDEX -- A -- B -- C -- D -- E -- F -- G -- H -- I -- K -- L -- M -- N -- P -- R -- S -- T -- U -- V -- W -- Z.

AbstractPreventing climate change and damage from natural disasters typically requires policies with up‐front costs that promise a flow of benefits over time. Why has obtaining such policies in a competitive electoral democracy proved so intractable? We develop a formal model of electoral accountability in this context, in which politicians have private information about their motivations. The model shows why fully rational voters, though certain that incumbents spend less on disaster prevention than is good for them, reelect incumbents at very high rates. In addition, in such equilibria, voters would punish incumbents who spent more on disaster prevention. This equilibrium is consistent with (and implies) some of the major empirical regularities observed in the literature on voting and disaster prevention. We discuss some implications of our analysis for advancing public debates about disaster and climate change mitigation.

This book provides a concise and up-to-date account of crime prevention theory, practice and research in a form designed to be accessible and interesting to both students and practitioners. Readers will be equipped to think in an informed and critical way about what has been and might be done in practice to prevent crime at local and national levels. What is distinctive in the approach is the emphasis on crime reduction mechanisms, how they may be activated and the intended and unintended patterns of outcome produced. Each of chapters two to five takes this as its organizing principle. The key

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This program was designed to inform about the dangerous and severe consequences when alcohol is misused in Texas.

Vaccinia virus (VACV) encodes the soluble type I interferon (IFN) binding protein B18 that is secreted from infected cells and also attaches to the cell surface, as an immunomodulatory strategy to inhibit the host IFN response. By using next generation sequencing technologies, we performed a detailed RNA-seq study to dissect at the transcriptional level the modulation of the IFN based host response by VACV and B18. Transcriptome profiling of L929 cells after incubation with purified recombinant B18 protein showed that attachment of B18 to the cell surface does not trigger cell signalling leading to transcriptional activation. Consistent with its ability to bind type I IFN, B18 completely inhibited the IFN-mediated modulation of host gene expression. Addition of UV-inactivated virus particles to cell cultures altered the expression of a set of 53 cellular genes, including genes involved in innate immunity. Differential gene expression analyses of cells infected with replication competent VACV identified the activation of a broad range of host genes involved in multiple cellular pathways. Interestingly, we did not detect an IFN-mediated response among the transcriptional changes induced by VACV, even after the addition of IFN to cells infected with a mutant VACV lacking B18. This is consistent with additional viral mechanisms acting at different levels to block IFN responses during VACV infection. ; Ministry of Economy and Competitiveness and European Union ; Peer Reviewed